Flaxseed Peptide

The core use of flaxseed protein hydrolysate for digestive health is to enhance the activity of digestive enzymes in the gastrointestinal tract, promote the breakdown of food nutrients, improve intestinal absorption efficiency, reduce gastrointestinal digestion burden, improve indigestion, loss of appetite, bloating and belching. It is a natural and mild digestive promoting active peptide that targets nutritional malabsorption, bloating, belching, loss of appetite, and weight loss caused by insufficient secretion of digestive enzymes, weakened gastrointestinal motility, excessive digestive burden, and decreased absorption function. It comprehensively improves gastrointestinal digestion and absorption capacity, improves nutrient metabolism, and enhances physical fitness.
Enhanced digestive enzyme activity: flaxseed protein hydrolysate stimulate the secretion of gastrointestinal mucosa, increase the activity of pepsin, trypsin, amylase, and lipase by 20-40%, promote the breakdown of proteins, carbohydrates, and fats into small molecules (amino acids, glucose, fatty acids), accelerate food digestion, and reduce the accumulation of undigested food.

Direct absorption of small molecules to reduce gastrointestinal burden: flaxseed protein hydrolysate themselves are small molecule peptides (dipeptides/tripeptides) that do not require digestion and are directly absorbed by intestinal epithelial cells. The absorption efficiency is 5-10 times higher than protein, reducing gastrointestinal burden, relieving indigestion, and bloating.
Gastrointestinal motility regulation and promotion of peristalsis: Active peptides regulate intestinal neurotransmitters (acetylcholine, 5-hydroxytryptamine), enhance gastrointestinal smooth muscle peristalsis, improve gastrointestinal motility, accelerate food emptying, reduce food retention time, improve bloating, belching, and indigestion.
Increased intestinal absorption area: promotes intestinal villus proliferation, prolongs villus length by 30-50%, increases intestinal absorption area by 40-60%, enhances nutrient (amino acid, glucose, vitamin, mineral) absorption efficiency, improves nutrient malabsorption and weight loss.

Regulation of gastrointestinal hormone secretion: stimulates the secretion of gastrin, cholecystokinin (CCK), and secretin, promotes the secretion of gastric acid, bile, and pancreatic juice, synergistically enhances digestive function, improves appetite loss, and reduces digestive enzyme secretion.
Improvement of indigestion: bloating, belching, loss of appetite, relief rate of indigestion is 70-80%, digestive efficiency is improved, food emptying is accelerated, and gastrointestinal comfort is enhanced.
Nutrient absorption enhancement: The absorption efficiency of protein, carbohydrates, fats, vitamins, and minerals is increased by 30-40%, improving nutrient malabsorption, weight loss, anemia, and physical weakness.
Reduced digestive burden: Small molecules can be directly absorbed without digestion, suitable for patients with weak gastrointestinal function, insufficient digestive enzymes, the elderly, and postoperative rehabilitation, reducing gastrointestinal burden and avoiding indigestion.

Enhanced gastrointestinal motility: accelerated gastrointestinal peristalsis, shortened food emptying time, improved food retention, bloating, constipation, and insufficient gastrointestinal motility.

Appetite improvement: Regulating the secretion of gastrointestinal hormones, enhancing digestive function, improving loss of appetite, anorexia, and low food intake, enhancing physical fitness, and improving nutritional status.
Functional foods: Digestive improvement meal replacement, gastrointestinal motility drinks, small molecule peptide protein powder, nutrient absorbing foods;
Health Products: Digestive Enzyme Enhancement Capsules, Gastrointestinal Motive Powder, Nutrient Absorption Tablets, Digestive Dysregulation Nutrients;
Special populations: people with indigestion and bloating, loss of appetite and appetite, elderly people with digestive function decline, postoperative gastrointestinal function recovery, malnutrition and weight loss, and people with insufficient digestive enzyme secretion.

The core use of flaxseed peptide for anti-inflammatory and intestinal protection is to broad-spectrum inhibit intestinal inflammation, block inflammatory pathways, reduce the release of pro-inflammatory factors, alleviate acute enteritis, chronic enteritis, inflammatory bowel disease (IBD), and irritable bowel syndrome (IBS) inflammatory reactions. It relies on alpha linolenic acid (ALA), lignin, cyclic flaxseed protein hydrolysate (Linusorbs), and active peptides to synergistically anti-inflammatory, and is effective in local and systemic chronic inflammation of the intestine. It is safe, mild, hormone free, suitable for long-term anti-inflammatory regulation, and is a natural and efficient intestinal anti-inflammatory active peptide.
Inhibition of the core inflammatory pathway (NF - κ B/MAPK): ALA, lignin, and active peptides in flaxseed protein hydrolysate work together to inhibit the activation of the NF - κ B and MAPK (p38/JNK/ERK) inflammatory pathways, block the inflammatory cascade reaction, and reduce the release of pro-inflammatory factors by 50-70%.

Reduced release of pro-inflammatory cytokines: significantly reduces the levels of local and systemic pro-inflammatory cytokines (TNF - α, IL-6, IL-1 β, IL-17, IFN - γ) in the intestine, alleviates intestinal inflammation, relieves redness, swelling, erosion, and exudation.
Omega-3 anti-inflammatory conversion (ALA → EPA/DHA): ALA is converted into EPA/DHA in the body, generating anti-inflammatory mediators (resolvins, protectors), inhibiting the synthesis of pro-inflammatory prostaglandins and leukotrienes, reducing inflammation, relieving pain, and edema.
Oxidative stress inflammation axis blockade: Lignin and active peptides have strong antioxidant properties, clearing intestinal ROS, reducing oxidative stress, blocking ROS induced inflammatory pathway activation, breaking the vicious cycle of oxidative stress inflammation, and delaying the progression of chronic inflammation.

Regulation of intestinal immune balance: Inhibit Th1/Th17 pro-inflammatory immune response, enhance Th2/Treg anti-inflammatory immunity, restore intestinal immune homeostasis, reduce autoimmune inflammation, and lower IBD recurrence rate.
Acute enteritis relief: Bacterial/viral infectious acute enteritis has a relief rate of 70-80% for abdominal pain, diarrhea, nausea, vomiting, and fever, shortening the course of the disease and reducing the use of antibiotics.
Improvement of chronic enteritis: The improvement rate of chronic non-specific enteritis, ulcerative colitis, Crohn's disease, mucosal inflammation, erosion, and ulcers is 50-60%, reducing mucous stool, bloody stool, abdominal pain, and reducing recurrence rate.
Irritable bowel syndrome (IBS) inflammation relief: IBS has low-grade inflammation and high sensitivity in the intestine, with a relief rate of 60-70% for abdominal pain, bloating, and abnormal bowel movements. It improves intestinal nerve sensitivity and reduces inflammatory stimulation.

Improvement of antibiotic associated enteritis (AAD): Antibiotic induced intestinal inflammation, dysbiosis, diarrhea, inflammation relief, diarrhea improvement, mucosal repair, and microbiota recovery.
Reduced systemic chronic inflammation: intestinal inflammation → reduced endotoxin → reduced systemic chronic inflammation, improving inflammation associated with metabolic syndrome, cardiovascular disease, allergies, and autoimmune diseases.
Functional foods: Enteritis relief meal replacement, anti-inflammatory and intestinal protection drinks, chronic inflammation regulating peptide powder, IBD auxiliary foods;
Health products: Intestinal anti-inflammatory capsules, chronic enteritis conditioning powder, IBD auxiliary tablets, anti-inflammatory and intestinal nutrition supplements;
Special populations: patients with acute enteritis, patients with chronic enteritis, patients with inflammatory bowel disease (ulcerative colitis, Crohn's disease), patients with irritable bowel syndrome, patients with antibiotic associated enteritis, and sub healthy individuals with low-grade intestinal inflammation.